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Barrett MT, Pritchard D, Palanca-Wessels C, Anderson j, Reid BJ and Rabinovitch PS Molecular Phenotype of Spontaneously Arising 4N (G2-Tetraploid) Intermediates of Neoplastic Progression in Barrett's Esophagus. Cancer Research 2003 63: p. 4211-17 |
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Barrett MT, Reid BJ, and Joslyn G Genotypic analysis of multiple loci in somatic cells by whole genome amplification. Nucleic Acids Research 1995 23(17): p. 3488-92 |
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Barrett MT, Sanchez CA, Prevo LJ, Wong DJ, Galipeau PC, Paulson TG, Rabinovitch PS, and Reid BJ Evolution of neoplastic cell lineages in Barrett's oesophagus. Nature Genetics 1999 22(1): p. 106-9 |
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Galipeau PC, Cowan DS, Sanchez CA, Barrett MT, Emond MJ, Levine DS, Rabinovitch PS, and Reid BJ 17p (p53) allelic losses, 4N (G2/tetraploid) populations, and progression to aneuploidy in Barrett's esophagus. Proceedings of the National Academy of Sciences of the United States of America 1996 93(14): p. 7081-4 |
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Galipeau PC, Prevo LJ, Sanchez CA, Longton GM, and Reid BJ Clonal expansion and loss of heterozygosity at chromosomes 9p and 17p in premalignant esophageal (Barrett's) tissue. Journal of the National Cancer Institute 1999 91(24): p. 2087-95 |
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Maley CC, Galipeau PC, Li X, Sanchez CA, Paulson TG and Reid BJ The Combination of Genetic Instability and Clonal Expansion Predicts Progression to Esophageal Adenocarcinoma. Cancer Research 2004 64: p. 3414-27 |
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Maley CC, Galipeau PC, Li X, Sanchez CA, Paulson TG, Blount PL and Reid BJ Selectively Advantageous Mutations and Hitchhikers in Neoplasms: p16 Lesions Are Selected in Barrett’s Esophagus. Cancer Research 2004 64: p. 7629-33 |
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Maley CC, Reid BJ and Forrest S Cancer Prevention Strategies That Address the Evolutionary Dynamics of Neoplastic Cells: Simulating Benign Cell Boosters and Selection for Chemosensitivity. Cancer Epidemiology, Biomarkers and Prevention 2004 13(8): p. 1375-84 |
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Mei R, Galipeau PC, Prass C, Berno A, Ghandour G, Patil N, Wolff RK, Chee MS, Reid BJ, and Lockhart DJ Genome-wide Detection of Allelic Imbalance Using Human SNPs and High Density DNA Arrays Genome Research 2000 10: p 1126-37 |
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Palanca-Wessels C, Klinglhutz A, Reid BJ, Norwood TH, Opheim KE, Paulson TG and Feng Z Extended lifespan of Barrett's esophagus epithelium transduced with the human telomerase catalytic subunit: a useful in vitro model. Carcinogenesis 2003 24(7): p. 1183-90 |
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Paulson TG, Galipeau PC, and Reid BJ Loss of heterozygosity analysis using whole genome amplification, cell sorting, and fluorescence-based PCR. Genome Research 1999 9(5): p. 482-91 |
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Prevo LJ, Sanchez CA, Galipeau PC, and Reid BJ p53 Mutant Clones and Field Effects in Barrett's Esophagus Cancer Research 1999 59: p. 4784-7 |
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Rabinovitch PS, Longton G, Blount PL, Levine DS, and Reid BJ Predictors of progression in Barrett's esophagus III: baseline flow cytometric variables. American Journal of Gastroenterology 2001 96(11): p. 3071-83 |
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Reid BJ, Levine DS, Longton G, Blount PL, and Rabinovitch PS Predictors of Progression to Cancer in Barrett's Esophagus: Baseline Histology and Flow Cytometry Identify Low- and High-Risk Patient Subsets American Journal of Gastroenterology 1999 95(7): p.1669 |
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Reid BJ, Prevo LJ, Galipeau PC, Sanchez CA, Longton G, Levine DS, Blount PL, and Rabinovitch PS Predictors of Progression in Barrett's Esophagus II: Baseline 17p (p53) Loss of Heterozygosity Identifies a Patient Subset at Increased Risk for Neoplastic Progression. American Journal of Gastroenterology 2001 96: p. 2839-48 |
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Rudolph RE, Vaughan TL, Kristal AR, Blount PL, Levine DS, Galipeau PC, Prevo LJ, Sanchez CA, Rabinovitch PS, and Reid BJ Serum Selenium Levels in Relation to Markers of Neoplastic Progression Among Persons With Barrett's Esophagus. JNCI 2003 95(10): p. 750-7 |
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Vaughan TL, Kristal AR, Blount PL, Levine DS, Galipeau PC, Prevo LJ, Sanchez CA, PS R, and BJ R NSAID use, BMI, and anthropometry in relation to genetic and flow cytometric abnormalities in Barrett's Esophagus Cancer Epidemiology, Biomarkers and Prevention 2002 11: p. 745-52 |
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Wong DJ, Paulson TG, Prevo LJ, Galipeau PC, Longton G, Blount PL, and Reid B J p16 INK4a lesions are common, early abnormalities that undergo clonal expansion in Barrett's metaplastic epithelium. Cancer Research 2001 61: p. 8284-9 |
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